Leptin receptors are expressed in the arcuate nucleus and dorsomedial hypothalamus. In a study episodic migraineurs showed a similar trend toward higher levels of total and high molecular weight adiponectin though neither measure reached statistical significance [ 61 ]. Data achieved through familial hemiplegic migraine suggest that; mutations in channel proteins can lead dysregulation of calcium and sodium levels and hyperexcitability in nervous system. Inflammation molecules involved in the pathogenesis of migraine is shown in Table 1.
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Migraine and Obesity: Possible Link to Inflammation
Even though, genetic tendencies can be responsible does being fat cause sex headaches migraine, accumulating data show that many metabolic or environmental factors can also trigger migraine attacks and its severity. As conflicting results of the studies have hindered any definite conclusion neither about a possible increased risk of having migraine for obese subjects nor a possible increased risk of being obese in the presence of migraine, meta-analysis were recoursed. All Published work is licensed under a Creative Commons Attribution 4. The extracranial terminal branches of the external carotid artery were suspected to be the reason of pain in the aura [ 1112 ].
The first approaches had defined migraine as a vascular disease and a phenomenon of intracranial vasospasm in cerebral arteries, causing vasodilation. In pediatric population Hershey et al. LMW ratio of adiponectin was suggested to be potential novel biomarkers for episodic migraine and either antagonist of high molecular weight adiponectin or agonists of low molecular weight adiponectin, or both suggested to be potential targets for headache management.
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